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Actinomycetes are widely distributed in nature, in particular on plants, they can enter the body with plants and be on the mucous membranes as a saprophyte. The transition of actinomycetes from a saprophytic to a parasitic state is facilitated by inflammatory diseases of the mucous membranes of the oral cavity, respiratory and gastrointestinal tract. At the site of the introduction of cenforce, an infectious granuloma is formed, which grows into the surrounding tissues. Abscesses appear in the granulations, which, breaking through, form fistulas. Skin lesions are secondary.

In the formation of suppuration, a secondary, mainly staphylococcal infection also plays a role. Antigens of radiant fungi lead to specific sensitization and allergic restructuring of the body (hypersensitization of the delayed or cenforce type), as well as to the formation of antibodies (complement binding, agglutinins, precipitins, etc.).

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The duration of the incubation period is unknown. It can vary widely and reach up to several years (from the time of infection to the development of manifest forms of actinomycosis). The main clinical forms of actinomycosis: actinomycosis of the head, tongue and neck; thoracic actinomycosis; abdominal; actinomycosis of the genitourinary organs; actinomycosis of cenforce; mycetoma (Madura foot); actinomycosis of the central nervous system. Actinomycosis refers to primary chronic infections with a long progressive course. With the growth of the infiltrate, the skin is involved in the process. Initially, a very dense and almost painless infiltrate is determined, the skin becomes cyanotic-purple, a fluctuation appears, and then long-term non-healing fistulas develop. In the pus, whitish-yellowish small lumps (drusen) are found.

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